[1] Medzhitov R. Approaching the asymptote:20 years later[J]. Immunity, 2009, 30(6):766-775.
[2] Iwasaki A, Medzhitov R. Toll-like receptor control of the adaptive immune responses[J]. Na Immunol, 2004, 5(10):987-995.
[3] Brown GD. Dectin-1:a signalling non-TLR pattern-recognition receptor[J]. Nat Rev Immunol, 2006, 6(1):33.
[4] Ichinohe T, Pang I K, Iwasaki A. Influenza virus activates inflammasomes via its intracellular M2 ion channel[J]. Nat Immunol, 2010, 11(5):404-410.
[5] Brubaker SW, Bonham KS, Zanoni I, et al. Innate immune pattern recognition:a cell biological perspective[J]. Annu Rev Immunol, 2015, 33(1):257-290.
[6] Uematsu S,Takeuchi O. Pathogen recognition and innate immunity[J]. Cell,2006,124(4):783-801.
[7] Strowig T, Henao-Mejia J, Elinav E, et al. Inflammasomes in health and disease[J]. NATURE, 2012, 481(7381):278-286.
[8] Gross O, Poeck H, Bscheider M, et al. Syk kinase signalling couples to the Nlrp3 inflammasome for anti-fungal host defence[J]. Nature, 2009, 459(7245):433.
[9] 吴丹. 炎症小体调控机制的研究进展[J]. 微生物学免疫学进展, 2015, 43(1):59-63.
[10] Schroder K, Tschopp J. The inflammasomes[J]. Cell, 2010, 140(6):821-832.
[11] Zhou R, Yazdi AS, Menu P, et al. A role for mitochondria in NLRP3 inflammasome activation[J]. Nature, 2011, 475(7354):122.
[12] Dostert C, Petrilli V, Van Bruggen R, et al. Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica[J]. Science, 2008, 320(5876):674-677.
[13] Cruz CM, Rinna A, Forman HJ, et al. ATP activates a reactive oxygen species-dependent oxidative stress response and secretion of proinflammatory cytokines in macrophages[J]. J Biol Chem, 2007, 282(5):2871-2879.
[14] Zhou R, Yazdi AS, Menu P, et al. A role for mitochondria in NLRP3 inflammasome activation[J]. Nature, 2011, 475(7354):122.
[15] Pétrilli, V, Papin S, Dostert C, et al. Activation of the NALP3 inflammasome is triggered by low intracellular potassium concentration[J]. Cell Death Differ, 2007, 14(9):1583-1589.
[16] Pelegrin P, Surprenant A. Pannexin-1 mediates large pore formation and interleukin-1beta release by the ATP-gated P2X7 receptor[J]. Embo J, 2014, 25(21):5071-5082.
[17] 王岩, 孙兵. 炎症小体及其相关疾病的研究进展[J]. 中国免疫学杂志, 2015,31(6):721-727.
[18] Hornung V, Bauernfeind FG, Halle A, et al. Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization[J]. Nature Immunology, 2008, 9(8):847-856.
[19] Cassel SL, Joly S, Sutterwala FS. The NLRP3 inflammasome:a sensor of immune danger signals[J]. Semin Immunol, 2009, 21(4):194-198.
[20] Li H, Wu S, Mao L.Human pathogenic fungus Trichophyton schoenleinii activates the NLRP3 inflammasome[J]. Protein Cell, 2013, 4(7):529-538.
[21] 帕丽达·阿布利孜,Fukushima K,邓淑文,等.新疆地区许兰毛癣菌的PCR-RAPD分子分型研究[J]. 中华皮肤科杂志, 2007, 40(8):499-500.
[22] Kankkunen P,Teirila L,Rintahaka J,et al.(1,3)-beta-glucans activate both decin-1 and NLRP3inflammasome in human macrophages[J].J Immunol 2010,184(11):6335-6342.
[23] Mao L, Zhang L, Li H, et al. Pathogenic fungus Microsporum canis activates the NLRP3 inflammasome[J]. Infect Immun, 2014, 82(2):882-892.
[24] 张丽萍, 帕丽达·阿布力孜. 炎症小体在真菌感染中的作用机理研究进展[J]. 中国美容医学杂志, 2014, 23(11):941-945.
[25] François L, Mayer, Wilson D, Hube B. Candida albicans pathogenicity mechanisms[J]. Virulence, 2013, 4(2):10.
[26] Wenzel R, Del Favero A, Kibbler C,et al. Economic evaluation of voriconazole compared with conventional amphotericin B for the primary treatment of aspergillosis in immunocompromised patients[J]. J Antimicrob Chemother, 2005, 55(3):352-361.
[27] Hoang LMN, Maguire JA, Doyle P, et al. Cryptococcus neoformans infections at Vancouver Hospital and Health Sciences Centre (1997-2002):epidemiology, microbiology and histopathology[J]. J Med Microbiol, 2004, 53(9):935-940.
[28] Joly S, Ma N, Sadler JJ, et al. Cutting edge:Candida albicans hyphae formation triggers activation of the NlrP3 inflammasome[J]. J Immunol, 2010, 183(4):3578-3581.
[29] Lee P, Lee DJ, Chan C, et al. Dynamic expression of epidermal caspase 8 simulates a wound healing response[J]. Nature, 2009, 458(7237):519-523.
[30] Robertson EJ, Wolf JM, Casadevall A. EDTA inhibits biofilm formation, extracellular vesicular secretion, and shedding of the capsular polysaccharide glucuronoxylomannan by Cryptococcus neoformans[J]. Appl Environ Microbiol, 2012, 78(22):7977-7984.
[31] Giri S, Kindo AJ. A review of Candida species causing blood stream infection[J]. Indian J Med Microbiol, 2012, 30(3):270-278.
[32] Silva NC. Aspartic proteinases of Candida spp.:role in pathogenicity and antifungal resistance[J]. Mycoses, 2013, 57(1):1-11.
[33] Braga-Silva LA, Santos ALS. Aspartic protease inhibitors as potential anti-Candida albicans drugs:impacts on fungal biology, virulence and pathogenesis[J]. Curr Med Chem, 2011, 18(16):2401-2419.
[34] Pietrella D, Pandey N, Gabrielli E, et al. Secreted aspartic proteases of Candida albicans activate the NLRP3 inflammasome[J]. Eur J Immunol, 2013, 43(3):679-692.
[35] 徐志庆, 吴大强, 邵菁, 等. NLRP3炎症小体与真菌感染[J]. 中国真菌学杂志, 2017,12(3):162-167.
[36] SaïdSadier N, Padilla E, Langsley G, et al. Aspergillus fumigatus stimulates the NLRP3 inflammasome through a pathway requiring ROS production and the Syk tyrosine kinase[J]. Plos One, 2010, 5(4):e10008.
[37] 江文俊, 姜福全, 崔彦. 白色念珠菌生物学特性研究进展[J]? 基础医学与临床, 2014, 34(4):550-554.
[38] Claudia F, Loures Flávio V, Frank de Araújo Eliseu, et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection[J]. PLoS ONE, 2013, 8(1):e54845.
[39] Karki R, Man S, Malireddi RK, et al. Concerted activation of the AIM2 and NLRP3 inflammasomes orchestrates host protection against Aspergillus infection[J]. Cell Host Microbe, 2015, 17(3):357-368.
[40] Ruiz A, Fromtling RA, Bulmer GS. Distribution of Cryptococcus neoformans in a natural site[J]. Infect Immun, 1981, 31(2):560.
[41] Velagapudi R,Hsueh YP, Geunes-Boyer S,et al. Spores as infectious propagules of Cryptococcus neoformans res as infectious propagules of Cryptococcus neoformans[J]. Infec Immun,2009,77(10):4345-4355.
[42] Zaragoza O, Rodrigues ML, De Jesus M, et al. The capsule of the fungal pathogen Cryptococcus neoformans[J]. Adv Appl Microbiol, 2009, 68(1):133-216.
[43] Guo C,Chen M,Fa Z,et al. Acapsular Cryptococcus neoformans activates the NLRP3 inflammasome[J]. Microbes Infect, 2014, 16(10):845-854.
[44] Lamkanfi M, Mueller JL, Vitari AC, et al. Glyburide inhibits the Cryopyrin/Nalp3 inflammasome[J]. J Cell Biol, 2009, 187(1):61-70.
[45] 孟广勋, 郭彩琴, 陈明宽. 天然免疫系统与新生隐球菌感染[J]. 中国真菌学杂志, 2015, 10(4):193-196.