[1] Ostrosky-Zeichner L, Rex JH, Bennett J, et al.Deeply invasive candidiasis[J].Infect Dis Clin North Am, 2002, 6(4):821-835.
[2] Cuéllar-Cruz M, Vega-González A, Mendoza-Novelo B, et al.The effect of biomaterials andantifungals onbiofilm formation by Candida species:a review[J].Eur J Clin Microbiol Infect Dis, 2012, 31(10):2513-2527.
[3] Chandra J, Kuhn DM, Mukherjee PK, et al.Biofilm formation by the fungal pathogen Candida albicans:development, architecture, anddrug resistance[J].J Bacteriol, 2001, 183(18):5385-5394.
[4] Michalek SM, Moore RN, McGhee JR, et al.The primaryrole of lymphoreticular cells in the mediation of host responses to bacterialendotoxim[J].J Infect Dis, 1980, 141(1):55-63.
[5] Morrison DC, Ryan JL.Endotoxins and disease mechanisms[J].Annu Rev Med., 1987, 38(1):417-432.
[6] Henry-Stanley MJ, Hess DJ, Erickson EA, et al.Effect of lipopolysaccharide on virulence of intestinal Candida albicans[J].J Surg Res, 2003, 113(1):42-49.
[7] Burd RS, Raymond CS, Dunn DL.Endotoxin promotes synergistic lethality duringconcurrent Escherichia coli and Candida albicans infection[J].J Surg Res, 1992, 52(6):537-542.
[8] Chandra J, Kuhn DM, Mukherjee PK, et al.Biofilm formation by the fungal pathogen Candida albicans:development, architecture, anddrug resistance[J].J Bacteriol, 2001, 183(18):5385-5394.
[9] Kerr JR.Suppression of fungal growth exhibited by Pseudomonas aeruginosa[J].J Clin Microbiol, 1994, 32(2):525-527.
[10] Gupta N, Haque A, Mukhopadhyay G, et al.Interactions between bacteria and Candida in the burn wound.[J].Burns, 2005, 31(3):375-378.
[11] Azoulay E, Timsit JF, Tafflet M, et al.Candida colonization of the respiratory tract and subsequent pseudomonas ventilator-associated pneumonia[J].Chest, 2006, 129(1):110-117.
[12] Sabra W, Lünsdorf H, Zeng AP.Alterations in the formation of lipopolysaccharide and membrane vesicles on the surface of Pseudomonas aeruginosa PAO1 under oxygen stressconditions[J].Microbiology, 2003, 149(Pt10):2789-2795.
[13] Bandara HM, K Cheung BP, Watt RM, et al.Pseudomonas aeruginosa lipopolysaccharide inhibits Candida albicans hyphae formation andalters gene expression during biofilm development[J].Mol Oral Microbiol, 2013, 28(1):54-69.
[14] Yeater KM, Chandra J, Cheng G, et al.Temporal analysis of Candida albicans gene expression during biofilm development[J].Microbiology, 2007, 153(Pt 8):2373-2385.