[1] JENKS J D, CORNELY O A, CHEN S C, et al.Breakthrough invasive fungal infections:Who is at risk[J]?Mycoses, 2020, 63(10):1021-1032. [2] SAKAGAMI T, KAWANO T, YAMASHITA K, et al.Antifungal susceptibility trend and analysis of resistance mechanism for Candida species isolated from bloodstream at a Japanese university hospital[J].Infect Chemother, 2019, 25(1):34-40. [3] HARRIS S D.Cdc42/Rho GTPases in fungi:variations on a common theme[J].Molecular Microbiology, 2011, 79(5):1123-1127. [4] BECKER J M, KAUFFMAN S J, HAUSER M, et al.Pathway analysis of Candida albicans survival and virulence determinants in a murine infection model[J].Proc Natl Acad Sci U S A, 2010, 107(51):22044-22049. [5] SMITH S E, CSANK C, REYES G, et al.Candida albicansRHO1 is required for cell viability in vitro and in vivo[J].FEMS Yeast Res, 2002, 2(2):103-111. [6] KONDOH O, TACHIBANA Y, OHYA Y, et al.Cloning of the RHO1 gene from Candida albicans and its regulation of beta-1, 3-glucan synthesis[J].Bacteriol, 1997, 179(24):7734-7741. [7] VANDENBERG A L, IBRAHIM A S, EDWARDS J E Jr, et al.Cdc42p GTPase regulates the budded-to-hyphal-form transition and expression of hypha-specific transcripts in Candida albicans[J].Eukaryot Cell, 2004, 3(3):724-734. [8] PEDRAZA N, CEMELI T, MONSERRAT M V, et al.Regulation of small GTPase activity by G1 cyclins[J].Small GTPases, 2019, 10(1):47-53. [9] BASSILANA M, ARKOWITZ R A.Rac1 and Cdc42 have different roles in Candida albicans development[J].Eukaryot Cell, 2006, 5(2):321-329. [10] AL-TAWFIQ J A, ALHUMAID S, ALSHUKAIRI A N, et al.COVID-19 and mucormycosis superinfection:the perfect storm[J].Infection, 2021, 49(5):833-853. |