[1] BUIL J B, HARE R K, ZWAAN B J, et al. The fading boundaries between patient and environmental routes of triazole resistance selection in Aspergillus fumigatus[J]. PLoS Pathog, 2019,15(8):e1007858. [2] SEGAL B H. Role of macrophages in host defense against Aspergillosis and strategies for immune augmentation[J]. Oncologist, 2007,12(Suppl 2):7-13. [3] HOGABOAM C M, TAKAHASHI K, EZEKOWITZ R A, et al. Mannose-binding lectin deficiency alters the development of fungal asthma:effects on airway response, inflammation, and cytokine profile[J]. J Leukoc Biol, 2004,75(5):805-814. [4] SERRANO-GOMEZ D, DOMINGUEZ-SOTO A, ANCOCHEA J, et al. Dendritic cell-specific intercellular adhesion molecule 3-grabbing nonintegrin mediates binding and internalization of Aspergillus fumigatus conidia by dendritic cells and macrophages[J]. J Immunol, 2004,173(9):5635-5643. [5] LUTHER K, TOROSANTUCCI A, BRAKHAGE A A, et al. Phagocytosis of Aspergillus fumigatus conidia by murine macrophages involves recognition by the dectin-1 beta-glucan receptor and Toll-like receptor 2[J]. Cell Microbiol, 2007,9(2):368-381. [6] LUTHER K, ROHDE M, STURM K, et al. Characterisation of the phagocytic uptake of Aspergillus fumigatus conidia by macrophages[J]. Microbes Infect, 2008,10(2):175-184. [7] 廖军, 钟白玉, 郝飞. 烟曲霉刺激下TLR4对大鼠肺泡巨噬细胞释放TNF-α和IL-1β作用[J]. 中国皮肤性病学杂志, 2008,22(6):328-330. [8] BROWN G D, HERRE J, WILLIAMS D L, et al. Dectin-1 mediates the biological effects of β-glucans[J]. J Exp Med, 2003,197(9):1119-1124. [9] 胡素侠, 杨小康, 何江. 烟曲霉感染时NADPH氧化酶通过诱导活性氧活化巨噬细胞自噬[J]. 细胞与分子免疫学杂志, 2015,31(2):190-193. [10] 龙南彪. 烟曲霉中调控胞内铁离子平衡的毒力相关蛋白LeuB和MrsA的功能研究[D]. 南京:南京师范大学, 2017. [11] CAFFREY A K, LEHMANN M M, ZICKOVICH J M, et al. IL-1α signaling is critical for leukocyte recruitment after pulmonary Aspergillus fumigatus challenge[J]. PLoS Pathog, 2015,11(1):e1004625. [12] WANG S, YUAN A, ZENG L, et al. The putative polysaccharide synthase AfCps1 regulates Aspergillus fumigatus morphogenesis and conidia immune response in mouse bone marrow-derived macrophages[J]. J Microbiol, 2021,59(1):64-75. [13] 易浔飞, 连兰兰, 兰小鹏. 烟曲霉致病性与免疫系统相互作用的研究进展[J]. 临床检验杂志, 2016,34(2):132-136. [14] 廖军. 肺泡巨噬细胞与烟曲霉相互作用的实验研究[D]. 重庆:第三军医大学, 2005. [15] WONG W, GOEHRING A S, KAPILOFF M S, et al. mAKAP compartmentalizes oxygen-dependent control of HIF-1α[J]. Sci Signal, 2008,1(51):a18. [16] MISHRA O P, RANDIS T, ASHRAF Q M, et al. Hypoxia-induced Bax and Bcl-2 protein expression, caspase-9 activation, DNA fragmentation, and lipid peroxidation in mitochondria of the cerebral cortex of newborn piglets:The role of nitric oxide[J]. Neuroscience, 2006,141(3):1339-1349. [17] DOSTERT C, PETRILLI V, VAN BRUGGEN R, et al. Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica[J]. Science, 2008,320(5876):674-677. [18] CRUZ C M, RINNA A, FORMAN H J, et al. ATP activates a reactive oxygen species-dependent oxidative stress response and secretion of proinflammatory cytokines in macrophages[J]. J Biol Chem, 2007,282(5):2871-2879. [19] ZHOU R, YAZDI A S, MENU P, et al. A role for mitochondria in NLRP3 inflammasome activation[J]. Nature, 2011,469(7329):221-225. [20] ZIMMERMANN K, OPITZ N, DEDIO J, et al. NOSTRIN:A protein modulating nitric oxide release and subcellular distribution of endothelial nitric oxide synthase[J]. Proc Natl Acad Sci U S A, 2002,99(26):17167-17172. [21] 陆军, 王新颖, 汤文浩. 谷氨酰胺对白细胞介素-1β刺激下大鼠肝细胞一氧化氮的产生及线粒体膜电位的影响[J]. 中华急诊医学杂志, 2009,18(1):47-50. |