[1] SALVUCCI E. Microbiome, holobiont and the net of life[J]. Crit Rev Microbiol, 2016, 42(3): 485-494.
[2] OH J, BYRD A L, PARDark M, et al. Temporal stability of the human skin microbiome[J]. Cell, 2016, 165(4): 854-866.
[3] GALLO R L. Human skin is the largest epithelial surface for interaction with microbes[J]. J Invest Dermatol, 2017, 137(6): 1213-1214.
[4] BYRD A L, BELKAID Y, SEGRE J A. The human skin microbiome[J]. Nat Rev Microbiol, 2018, 16(3): 143-155.
[5] TIPTON L, MULLER C L, KURTZ Z D, et al. Fungi stabilize connectivity in the lung and skin microbial ecosystems[J]. Microbiome, 2018, 6(1): 12.
[6] FINDLEY K, OH J, YANG J, et al. Topographic diversity of fungal and bacterial communities in human skin[J]. Nature, 2013, 498(7454): 367-370.
[7] JO J H, KENNEDY E A, KONG H H. Topographical and physiological differences of the skin mycobiome in health and disease[J]. Virulence, 2017, 8(3): 324-333.
[8] HARADA K, SAITO M, SUGITA T, et al. Malassezia species and their associated skin diseases[J]. J Dermatol, 2015, 42(3): 250-257.
[9] WHITE T C, FINDLEY K, DAWSON T L J R., et al. Fungi on the skin: dermatophytes and Malassezia[J]. Cold Spring Harb Perspect Med, 2014, 4(8): a019802.
[10] PROHIC A, JOVOVIC SADIKOVIC T, KRUPALIJA-FAZLIC M, et al. Malassezia species in healthy skin and in dermatological conditions[J]. Int J Dermatol, 2016, 55(5): 494-504.
[11] JO J H, DEMING C, KENNEDY E A, et al. Diverse human skin fungal communities in children converge in adulthood[J]. J Invest Dermatol, 2016, 136(12): 2356-2363.
[12] LIMON J J, SKALSKI J H, UNDERHILL D M. Commensal fungi in health and disease[J]. Cell Host Microbe, 2017, 22(2): 156-165.
[13] HAN S H, CHEON H I, HUR M S, et al. Analysis of the skin mycobiome in adult patients with atopic dermatitis[J]. Exp Dermatol, 2018, 27(4): 366-373.
[14] GLATZ M, BOSSHARD P P, HOETZENECKER W, et al. The Role of Malassezia spp. in Atopic Dermatitis[J]. J Clin Med, 2015, 4(6): 1217-1228.
[15] LUNJANI N, HLELA C, O'MAHONY L. Microbiome and skin biology[J]. Curr Opin Allergy Clin Immunol, 2019, 19(4): 328-333.
[16] YAN D, ISSA N, AFIFI L, et al. The role of the skin and gut microbiome in psoriatic disease[J]. Curr Dermatol Rep, 2017, 6(2): 94-103.
[17] SOARES R C, CAMARGO-PENNA P H, DE MORAES V C, et al. Dysbiotic bacterial and fungal communities not restricted to clinically affected skin sites in dandruff[J]. Front Cell Infect Microbiol, 2016, 6: 157. DOI: 10.3389/fcimb.2016.00157.
[18] WANG L, CLAVAUD C, BAR-HEN A, et al. Characterization of the major bacterial-fungal populations colonizing dandruff scalps in Shanghai, China, shows microbial disequilibrium[J]. Exp Dermatol, 2015, 24(5): 398-400.
[19] PIETRZAK A, GRYWALSKA E, SOCHA M, et al. Prevalence and possible role of Candida species in patients with psoriasis: a systematic review and meta-analysis[J]. Mediators Inflamm, 2018: 9602362. DOI: 10.1155/2018/9602362.
[20] DE JESUS-GIL C, SANS-DE SAN NICOLAS L, RUIZ-ROMEU E, et al. Interplay between humoral and CLA(+) T cell response against Candida albicans in psoriasis[J]. Int J Mol Sci, 2021, 22(4):1519.
[21] BOZENA D K, IWONA D, ILONA K. The mycobiome-a friendly cross-talk between fungal colonizers and their host[J]. Ann Parasitol, 2016, 62(3): 175-184.
[22] BURSTEIN V L, GUASCONI L, BECCACECE I, et al. IL-17-Mediated immunity controls skin infection and T helper 1 response during experimental Microsporum canis dermatophytosis[J] J Invest Dermatol, 2018, 138(8): 1744-1753.
[23] JOHANSSON H J, VALLHOV H, HOLM T, et al. Extracellular nanovesicles released from the commensal yeast Malassezia sympodialis are enriched in allergens and interact with cells in human skin[J]. Sci Rep, 2018, 8(1): 9182.
[24] RAYNER S, BRUHN S, VALLHOV H, et al. Identification of small RNAs in extracellular vesicles from the commensal yeast Malassezia sympodialis[J]. Sci Rep, 2017, 7: 39742. DOI: 10.1038/srep39742.
[25] SPARBER F, DE GREGORIO C, STECKHOLZER S, et al. The skin commensal yeast Malassezia triggers a type 17 response that coordinates anti-fungal immunity and exacerbates skin inflammation[J]. Cell Host Microbe, 2019, 25(3): 389-403,e386.
[26] PARK C O, FU X, JIANG X, et al. Staged development of long-lived T-cell receptor αβ T(H)17 resident memory T-cell population to Candida albicans after skin infection[J]. J Allergy Clin Immunol, 2018, 142(2): 647-662.
[27] USYK M, ZOLNIK C P, PATEL H, et al. Novel ITS1 fungal primers for characterization of the mycobiome[J]. mSphere, 2017, 2(6):e00488-17. |
